Preliminary experiments reveal conolidine could inhibit specific ion channels, lowering neuronal excitability and limiting ache alerts. This system is especially related in neuropathic ache, in which irregular signaling causes persistent pain. Furthermore, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to discomfort notion. Computational docking exper... https://barryv136pfy8.blogvivi.com/profile