Preliminary studies suggest conolidine may inhibit certain ion channels, lessening neuronal excitability and restricting suffering signals. This system is especially relevant in neuropathic ache, where by abnormal signaling causes persistent irritation. In addition, conolidine seems to impact G protein-coupled receptor (GPCR) pathways integral to agony notion. Computational docking sc... https://jinnahf368nld5.answerblogs.com/profile